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HLA-DOA
An Error has occured retrieving Wikidata item for infobox HLA-DOA (Major histocompatibility complex, class II, DO alpha) هوَ بروتين يُشَفر بواسطة جين HLA-DOA في الإنسان.[1][2]
الوظيفة
هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ "Entrez Gene: HLA-DOA major histocompatibility complex, class II, DO alpha". مؤرشف من الأصل في 2010-12-05.
- ^ Young JA، Trowsdale J (أغسطس 1990). "The HLA-DNA (DZA) gene is correctly expressed as a 1.1 kb mature mRNA transcript". Immunogenetics. ج. 31 ع. 5–6: 386–8. DOI:10.1007/BF02115015. PMID:2370084.
قراءة متعمقة
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- Rosenstein Y، Burakoff SJ، Herrmann SH (1990). "HIV-gp120 can block CD4-class II MHC-mediated adhesion". J. Immunol. ج. 144 ع. 2: 526–31. PMID:1967269.
- Bowman MR، MacFerrin KD، Schreiber SL، Burakoff SJ (1991). "Identification and structural analysis of residues in the V1 region of CD4 involved in interaction with human immunodeficiency virus envelope glycoprotein gp120 and class II major histocompatibility complex molecules". Proc. Natl. Acad. Sci. U.S.A. ج. 87 ع. 22: 9052–6. DOI:10.1073/pnas.87.22.9052. PMC:55099. PMID:1978941.
- Rosen-Bronson S، Long EO (1991). "An unusual form of alternative splicing in the HLA-DNA gene". Immunogenetics. ج. 33 ع. 2: 124–31. DOI:10.1007/BF00210826. PMID:1999352.
- Jonsson AK، Rask L (1989). "Human class II DNA and DOB genes display low sequence variability". Immunogenetics. ج. 29 ع. 6: 411–3. DOI:10.1007/BF00375872. PMID:2499532.
- Clayton LK، Sieh M، Pious DA، Reinherz EL (1989). "Identification of human CD4 residues affecting class II MHC versus HIV-1 gp120 binding". Nature. ج. 339 ع. 6225: 548–51. DOI:10.1038/339548a0. PMID:2543930.
- Diamond DC، Sleckman BP، Gregory T، وآخرون (1988). "Inhibition of CD4+ T cell function by the HIV envelope protein, gp120". J. Immunol. ج. 141 ع. 11: 3715–7. PMID:2846691.
- Inoko H، Ando A، Kimura M، Tsuji K (1985). "Isolation and characterization of the cDNA clone and genomic clones of a new HLA class II antigen heavy chain, DO alpha". J. Immunol. ج. 135 ع. 3: 2156–9. PMID:2991382.
- Trowsdale J، Kelly A (1986). "The human HLA class II alpha chain gene DZ alpha is distinct from genes in the DP, DQ and DR subregions". EMBO J. ج. 4 ع. 9: 2231–7. PMC:554490. PMID:3000765.
- Andrieu JM، Even P، Venet A (1986). "AIDS and related syndromes as a viral-induced autoimmune disease of the immune system: an anti-MHC II disorder. Therapeutic implications". AIDS research. ج. 2 ع. 3: 163–74. DOI:10.1089/aid.1.1986.2.163. PMID:3489470.
- Houlgatte R، Scarmato P، el Marhomy S، وآخرون (1994). "HLA class II antigens and the HIV envelope glycoprotein gp120 bind to the same face of CD4". J. Immunol. ج. 152 ع. 9: 4475–88. PMID:7512597.
- Chirmule N، McCloskey TW، Hu R، وآخرون (1995). "HIV gp120 inhibits T cell activation by interfering with expression of costimulatory molecules CD40 ligand and CD80 (B71)". J. Immunol. ج. 155 ع. 2: 917–24. PMID:7541827.
- Rowell JF، Stanhope PE، Siliciano RF (1995). "Endocytosis of endogenously synthesized HIV-1 envelope protein. Mechanism and role in processing for association with class II MHC". J. Immunol. ج. 155 ع. 1: 473–88. PMID:7602119.
- Chen YH، Böck G، Vornhagen R، وآخرون (1994). "HIV-1 gp41 binding proteins and antibodies to gp41 could inhibit enhancement of human Raji cell MHC class I and II expression by gp41". Mol. Immunol. ج. 31 ع. 13: 977–82. DOI:10.1016/0161-5890(94)90092-2. PMID:8084338.
- Chirmule N، Wang XP، Hu R، وآخرون (1994). "Envelope glycoproteins of HIV-1 interfere with T-cell-dependent B cell differentiation: role of CD4-MHC class II interaction in the effector phase of T cell help". Cell. Immunol. ج. 155 ع. 1: 169–82. DOI:10.1006/cimm.1994.1110. PMID:8168144.
- Callahan KM، Rowell JF، Soloski MJ، وآخرون (1993). "HIV-1 envelope protein is expressed on the surface of infected cells before its processing and presentation to class II-restricted T lymphocytes". J. Immunol. ج. 151 ع. 6: 2928–42. PMID:8376762.
- Liljedahl M، Kuwana T، Fung-Leung WP، وآخرون (1996). "HLA-DO is a lysosomal resident which requires association with HLA-DM for efficient intracellular transport". EMBO J. ج. 15 ع. 18: 4817–24. PMC:452218. PMID:8890155.
- Ruff AL، Guarnieri FG، Staveley-O'Carroll K، وآخرون (1997). "The enhanced immune response to the HIV gp160/LAMP chimeric gene product targeted to the lysosome membrane protein trafficking pathway". J. Biol. Chem. ج. 272 ع. 13: 8671–8. DOI:10.1074/jbc.272.13.8671. PMID:9079699.
- Douek DC، Altmann DM (1997). "HLA-DO is an intracellular class II molecule with distinctive thymic expression". Int. Immunol. ج. 9 ع. 3: 355–64. DOI:10.1093/intimm/9.3.355. PMID:9088974.