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TRA2B
An Error has occured retrieving Wikidata item for infobox TRA2B (Transformer 2 beta homolog) هوَ بروتين يُشَفر بواسطة جين TRA2B في الإنسان.[1][2]
الوظيفة
![[أيقونة]](/%D9%85%D9%84%D9%81:Crystal_xedit.svg){kind=small} | هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
| هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ "Entrez Gene: SFRS10 splicing factor, arginine/serine-rich 10 (transformer 2 homolog, Drosophila)". مؤرشف من الأصل في 2010-12-05.
- ^ Nayler O، Cap C، Stamm S (أكتوبر 1998). "Human transformer-2-beta gene (SFRS10): complete nucleotide sequence, chromosomal localization, and generation of a tissue-specific isoform". Genomics. ج. 53 ع. 2: 191–202. DOI:10.1006/geno.1998.5471. PMID:9790768.
قراءة متعمقة
- Matsuo N، Ogawa S، Imai Y، وآخرون (1996). "Cloning of a novel RNA binding polypeptide (RA301) induced by hypoxia/reoxygenation". J. Biol. Chem. ج. 270 ع. 47: 28216–22. DOI:10.1074/jbc.270.47.28216. PMID:7499316.
- Lennon G، Auffray C، Polymeropoulos M، Soares MB (1996). "The I.M.A.G.E. Consortium: an integrated molecular analysis of genomes and their expression". Genomics. ج. 33 ع. 1: 151–2. DOI:10.1006/geno.1996.0177. PMID:8617505.
- Beil B، Screaton G، Stamm S (1997). "Molecular cloning of htra2-beta-1 and htra2-beta-2, two human homologs of tra-2 generated by alternative splicing". DNA Cell Biol. ج. 16 ع. 6: 679–90. DOI:10.1089/dna.1997.16.679. PMID:9212162.
- Imai Y، Matsuo N، Ogawa S، وآخرون (1998). "Cloning of a gene, YT521, for a novel RNA splicing-related protein induced by hypoxia/reoxygenation". Brain Res. Mol. Brain Res. ج. 53 ع. 1–2: 33–40. DOI:10.1016/S0169-328X(97)00262-3. PMID:9473574.
- Tacke R، Tohyama M، Ogawa S، Manley JL (1998). "Human Tra2 proteins are sequence-specific activators of pre-mRNA splicing". Cell. ج. 93 ع. 1: 139–48. DOI:10.1016/S0092-8674(00)81153-8. PMID:9546399.
- Nayler O، Cap C، Stamm S (1998). "Human transformer-2-beta gene (SFRS10): complete nucleotide sequence, chromosomal localization, and generation of a tissue-specific isoform". Genomics. ج. 53 ع. 2: 191–202. DOI:10.1006/geno.1998.5471. PMID:9790768.
- Eldridge AG، Li Y، Sharp PA، Blencowe BJ (1999). "The SRm160/300 splicing coactivator is required for exon-enhancer function". Proc. Natl. Acad. Sci. U.S.A. ج. 96 ع. 11: 6125–30. Bibcode:1999PNAS...96.6125E. DOI:10.1073/pnas.96.11.6125. PMC:26846. PMID:10339552.
- Lai MC، Lin RI، Huang SY، وآخرون (2000). "A human importin-beta family protein, transportin-SR2, interacts with the phosphorylated RS domain of SR proteins". J. Biol. Chem. ج. 275 ع. 11: 7950–7. DOI:10.1074/jbc.275.11.7950. PMID:10713112.
- Venables JP، Elliott DJ، Makarova OV، وآخرون (2000). "RBMY, a probable human spermatogenesis factor, and other hnRNP G proteins interact with Tra2beta and affect splicing". Hum. Mol. Genet. ج. 9 ع. 5: 685–94. DOI:10.1093/hmg/9.5.685. PMID:10749975.
- Helmken C، Wirth B (2001). "Exclusion of Htra2-beta1, an up-regulator of full-length SMN2 transcript, as a modifying gene for spinal muscular atrophy". Hum. Genet. ج. 107 ع. 6: 554–8. DOI:10.1007/s004390000409. PMID:11153908.
- Young PJ، DiDonato CJ، Hu D، وآخرون (2002). "SRp30c-dependent stimulation of survival motor neuron (SMN) exon 7 inclusion is facilitated by a direct interaction with hTra2 beta 1". Hum. Mol. Genet. ج. 11 ع. 5: 577–87. DOI:10.1093/hmg/11.5.577. PMID:11875052.
- Hofmann Y، Wirth B (2003). "hnRNP-G promotes exon 7 inclusion of survival motor neuron (SMN) via direct interaction with Htra2-beta1". Hum. Mol. Genet. ج. 11 ع. 17: 2037–49. DOI:10.1093/hmg/11.17.2037. PMID:12165565.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. Bibcode:2002PNAS...9916899M. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Tran Q، Coleman TP، Roesser JR (2003). "Human transformer 2beta and SRp55 interact with a calcitonin-specific splice enhancer". Biochim. Biophys. Acta. ج. 1625 ع. 2: 141–52. DOI:10.1016/S0167-4781(02)00600-0. PMID:12531473.
- El Yazidi-Belkoura I، Adriaenssens E، Dollé L، وآخرون (2003). "Tumor necrosis factor receptor-associated death domain protein is involved in the neurotrophin receptor-mediated antiapoptotic activity of nerve growth factor in breast cancer cells". J. Biol. Chem. ج. 278 ع. 19: 16952–6. DOI:10.1074/jbc.M300631200. PMID:12604596.
- Jiang Z، Tang H، Havlioglu N، وآخرون (2003). "Mutations in tau gene exon 10 associated with FTDP-17 alter the activity of an exonic splicing enhancer to interact with Tra2 beta". J. Biol. Chem. ج. 278 ع. 21: 18997–9007. DOI:10.1074/jbc.M301800200. PMC:2140226. PMID:12649279.
- Chen X، Guo L، Lin W، Xu P (2004). "Expression of Tra2beta isoforms is developmentally regulated in a tissue- and temporal-specific pattern". Cell Biol. Int. ج. 27 ع. 6: 491–6. DOI:10.1016/S1065-6995(03)00072-6. PMID:12798777.
- Helmken C، Hofmann Y، Schoenen F، وآخرون (2004). "Evidence for a modifying pathway in SMA discordant families: reduced SMN level decreases the amount of its interacting partners and Htra2-beta1". Hum. Genet. ج. 114 ع. 1: 11–21. DOI:10.1007/s00439-003-1025-2. PMID:14520560.
- Li J، Hawkins IC، Harvey CD، وآخرون (2003). "Regulation of alternative splicing by SRrp86 and its interacting proteins". Mol. Cell. Biol. ج. 23 ع. 21: 7437–47. DOI:10.1128/MCB.23.21.7437-7447.2003. PMC:207616. PMID:14559993.
- Ota T، Suzuki Y، Nishikawa T، وآخرون (2004). "Complete sequencing and characterization of 21,243 full-length human cDNAs". Nat. Genet. ج. 36 ع. 1: 40–5. DOI:10.1038/ng1285. PMID:14702039.
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